The new antibiotic cubicin works by creating a passive transport channel for H+. Obviously this would affect the PMF, but other cell processes may be disrupted as well. How would treatment with cubicin affect flagellar motility and chemotaxis? Would there be a greater effect on the import of aspartate or of glucose via H+ symports? Explain. Would there be a greater effect on the import of the sugar maltose via a PTS system (group translocation) or via an ABC uptake system? Explain.